Using the data available, the results presented in
Table 1, were derived using the following steps in a
simple spreadsheet model:
! Prevalence data are reported in Section 2.3.1. The
probability of a positive sample, as a result, is
estimated to be 0.094 for Cryptosporidium, and
0.0201 for Giardia.
! In Section 2.3.2, it was reported that the concentration
of Cryptosporidium oocysts was between 2
and 6 per 100 g of sprouts, with a mean value of
approximately 3.4 oocysts/100 g. The concentration
of Giarda cysts was between 2 and 6 per 100
g of sprouts with a mean concentration of 4.0
cysts/100 g.
! Section 2.3.3 suggests that a typical mung bean
serving would be in the order of approximately 20
g. From estimates of the concentration per gram
and the serving size in grams, the dose per serving
can then be calculated:
o (#/gram)(gram/serving)=#/serving
! To translate the dose consumed to a risk of infection
or illness, a dose–response relationship is required.
In the case of microbial pathogens, the currently
accepted hypothesis is that for infection to succeed,
Using the data available, the results presented inTable 1, were derived using the following steps in asimple spreadsheet model:! Prevalence data are reported in Section 2.3.1. Theprobability of a positive sample, as a result, isestimated to be 0.094 for Cryptosporidium, and0.0201 for Giardia.! In Section 2.3.2, it was reported that the concentrationof Cryptosporidium oocysts was between 2and 6 per 100 g of sprouts, with a mean value ofapproximately 3.4 oocysts/100 g. The concentrationof Giarda cysts was between 2 and 6 per 100g of sprouts with a mean concentration of 4.0cysts/100 g.! Section 2.3.3 suggests that a typical mung beanserving would be in the order of approximately 20g. From estimates of the concentration per gramand the serving size in grams, the dose per servingcan then be calculated:o (#/gram)(gram/serving)=#/serving! To translate the dose consumed to a risk of infectionor illness, a dose–response relationship is required.In the case of microbial pathogens, the currentlyaccepted hypothesis is that for infection to succeed,
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