Bipolar I disorder (BD-I) patients have been often
reported as having alterations in neurocognitive
functioning, which appear during both acute stages
and euthymic periods (1–3). Such reductions in
cognitive function have been found mainly in
attention, executive function, and memory (4–9),
with significant psychomotor slowing also having
been observed (10). However, the affected cognitive
domains are not consistent in all studies (11, 12),
probably due to methodological differences and the
influence of clinical variables that could affect
cognitive performance.