As expected, the higher TBARS values were observed in the samples incorporated with encapsulated phosphates than the samples with unencapsulated counterparts at 24 h (p b 0.05).
This result showed that encapsulation prevented the antioxidant effect of phosphates in uncooked samples.
Furthermore, the higher (p b 0.05) TBARS were determined in beef than chicken in both uncooked and cooked samples (Table 2). This may be the result of higher iron level in beef.
Our results are also confirmed with the previous study of Min, Cordray, and Ahn (2010) reported that the increases of TBARS values in cooked beef loin pattieswere significantly higher than those of the cooked chicken breast patties with the same treatments.
The authors suggested that free ionic
iron is themajor catalyst for lipid oxidation, and the low“storage-stable and heat-stable” ferric ion reducing capacity in chicken breast were responsible for the high oxidative stability for raw and cooked chicken breast comparedwith beef loin under prooxidants, cooking, and storage conditions.
Faustman, Yin, and Nadeau (1992) reported that greater
concentrations of iron and myoglobin are associated with greater
rates of lipid oxidation. In addition, chicken meats are reported to contain
more histidine-containing dipeptides such as carnosine and anserine, which have antioxidant activities than beef and pork (Min, Nam,Cordray, & Ahn, 2008).
As expected, the higher TBARS values were observed in the samples incorporated with encapsulated phosphates than the samples with unencapsulated counterparts at 24 h (p b 0.05). This result showed that encapsulation prevented the antioxidant effect of phosphates in uncooked samples. Furthermore, the higher (p b 0.05) TBARS were determined in beef than chicken in both uncooked and cooked samples (Table 2). This may be the result of higher iron level in beef. Our results are also confirmed with the previous study of Min, Cordray, and Ahn (2010) reported that the increases of TBARS values in cooked beef loin pattieswere significantly higher than those of the cooked chicken breast patties with the same treatments. The authors suggested that free ioniciron is themajor catalyst for lipid oxidation, and the low“storage-stable and heat-stable” ferric ion reducing capacity in chicken breast were responsible for the high oxidative stability for raw and cooked chicken breast comparedwith beef loin under prooxidants, cooking, and storage conditions. Faustman, Yin, and Nadeau (1992) reported that greaterconcentrations of iron and myoglobin are associated with greaterrates of lipid oxidation. In addition, chicken meats are reported to containmore histidine-containing dipeptides such as carnosine and anserine, which have antioxidant activities than beef and pork (Min, Nam,Cordray, & Ahn, 2008).
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