The lead accumulation in kidney, liver, spleen, intestine, gill, and muscle of rockfish exposed to the dietary lead concentrations is demonstrated in Fig. 1. It was observed that the highest lead accumulation in kidney. A considerable increase was shown over 30 mg/kg at 2 and 4 weeks after the dietary lead exposure. The Pb accumulation values in kidney were 4.87±1.48 µg/g at 30 mg/kg, 8.02±1.02 µg/g at 60 mg/kg, 9.41±1.25 µg/g at 120 mg/kg, and 14.57±1.43 µg/g at 240 mg/kg dietary lead exposure after 2 weeks, and 6.24±1.27 µg/g at 30 mg/kg, 9.60±1.21 µg/g at 60 mg/kg, 12.89±1.62 µg/g at 120 mg/kg, and 17.11±1.52 µg/g at 240 mg/kg dietary lead exposure after 4 weeks. For the liver tissue, the Pb accumulation significantly increased over 30 mg/kg at 2 and 4 weeks, and the highest Pb accumulation value in liver was 14.61±1.64 µg/g for exposure to 240 mg/kg after 4 weeks. The Pb accumulation in spleen was also notably increased, reaching a value of 10.44±1.23 µg/g at dose for exposure to 240 mg/kg in 4 weeks. For the tissues of intestine and gill, significant accumulations was observed over 60 mg/kg, but the levels of Pb accumulation were much lower than the kidney, liver, and spleen. The highest Pb accumulation values were 5.72±0.72 µg/g for exposure to 240 mg/kg in intestine and 3.87±0.41 µg/g for exposure to 240 mg/kg in gill after 4 weeks. In the muscle Pb accumulation, a significant increase was observed at 240 mg/kg after 2 and 4 weeks. The Pb accumulation depended upon the dietary lead concentration and exposure periods. After 4 weeks of the dietary lead exposure, the profile of tissue Pb accumulation was kidney>liver>spleen>intestine>gill>muscle.