Conclusion
In conclusion, a continuous 3°C increase in the incubation temperature between ED 16 and ED 18.5 resulted in reduced embryo growth as well as a delay in the hatching process and increased embryonic mortality, which is linked to reduced thyroid hormone levels; decreased liver glycogen reserves, resulting in a decrease in the availability of blood glucose during the hatching process, hyperglycemia, or both in the days before hatch; and decreased plasma TG and NEFA levels. Although the hatching process of embryos exposed to lower incubation temperatures was also significantly retarded, they showed embryonic development and growth strikingly similar to those of the control temperature group, confirming that broiler embryos are more sensitive to high incubation temperatures than to low incubation temperatures (French, 1997).
ConclusionIn conclusion, a continuous 3°C increase in the incubation temperature between ED 16 and ED 18.5 resulted in reduced embryo growth as well as a delay in the hatching process and increased embryonic mortality, which is linked to reduced thyroid hormone levels; decreased liver glycogen reserves, resulting in a decrease in the availability of blood glucose during the hatching process, hyperglycemia, or both in the days before hatch; and decreased plasma TG and NEFA levels. Although the hatching process of embryos exposed to lower incubation temperatures was also significantly retarded, they showed embryonic development and growth strikingly similar to those of the control temperature group, confirming that broiler embryos are more sensitive to high incubation temperatures than to low incubation temperatures (French, 1997).
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