Regarding their pharmacokinetic properties and clinical success in the management of several cancer types, it is difficult to predict which type of RTK inhibitor will be successful in the suppression of development of endometriosis. To the best of our knowledge, there was one study used imatinib on the endometriotic tissue in a murine model to assess the role of being homozygous for a colony-stimulating factor 1 mutation on the rate of lesion formation.