One of the most striking results from the microarray study was the revelation that large numbers
of histones were downregulated in Fusarium-inoculated tissue as
a result of growth in epiBL-amended soil. Müssig et al.
found that, although BR did not significantly alter histone gene
expression in wild-type Arabidopsis plants, it inconsistently
downregulated His1-3 is Br-deficient dwf1-6 mutant plants. This
suggests that chromatin remodeling is either an effector of or
affected by epiBL signaling. This adds further weight to the
evidence that BR function requires chromatin remodeling .
Chromatin remodeling plays an important role in SA-mediated
systemic acquired resistance and SA has recently been
shown to play a role in FHB resistance . Chromatin remodeling
could be instrumental for the priming of epiBLresponsive
loci to respond to subsequent Fusarium spp. attack.