Fig. 5. Effect of CAE on the glutathione system in Ab1e40-treated cells. Differentiated PC12 (A, C) and IMR32 cells (B, D) were treated with aggregated Ab1e40 and CAE at indicated
concentrations for 24 h, and GR (A, B) and GPx (C, D) activities were measured. GR and GPx activities are displayed as a percentage of the control. (E) The GSH/GSSG ratio in
differentiated PC12 (-) and IMR32 (▫) cells were measured at 24 h after cotreatment with CAE and aggregated Ab1e40. In all experiments, cells left untreated served as controls.
Values are expressed as mean ± SD (n ¼ 6). *P < 0.05 compared to control, #P < 0.05 compared to Ab1e40 treatment alone.
Fig. 5. Effect of CAE on the glutathione system in Ab1e40-treated cells. Differentiated PC12 (A, C) and IMR32 cells (B, D) were treated with aggregated Ab1e40 and CAE at indicatedconcentrations for 24 h, and GR (A, B) and GPx (C, D) activities were measured. GR and GPx activities are displayed as a percentage of the control. (E) The GSH/GSSG ratio indifferentiated PC12 (-) and IMR32 (▫) cells were measured at 24 h after cotreatment with CAE and aggregated Ab1e40. In all experiments, cells left untreated served as controls.Values are expressed as mean ± SD (n ¼ 6). *P < 0.05 compared to control, #P < 0.05 compared to Ab1e40 treatment alone.
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