There was a strong significant positive relationship between
VCAM-1, ICAM-1, CRP and TXB2 with BP. This suggests
that elevation in the blood pressure in this study may be a consequence
of vascular inflammation as reflected by an increase
in VCAM-1, ICAM-1 and CRP in the circulation. The vascular
inflammation subsequently impaired production of TXB2
by vascular endothelium leading to an increase in the BP. In