The active compounds produced by Xenorhabdus spp. may act on
P. capsici and B. cinerea by different cellular action mechanisms. P.
capsici is an oomycete and differs from the fungi in cell wall composition
that contains or produce sterols43. The active compounds
xenocoumacins can act on P. capsici by reducing the activity of
transporters involved in transport systems of P. capsici cell membranes44.
B. cinerea is an ascomycete pathogen and spores of B.
cinerea are considered to be the main source of dispersal of inoculum,
and their germination and adhesion on plant surfaces represent crucial
steps preceding host penetration and colonization45,46