Thus, for our experiment, a model was chosen that included feeding a basal diet, which allowed for growth and the maintenance of digestive tract integrity, supplemented with infused energy in the form of carbohydrate. This model was used to test 2 separate hypotheses. The first hypothesis is that digestive tract and liver mass are responsive to energy input with the extent of the response dependent upon site of carbohydrate infusion. The second hypothesis concerns our previous calorimetric observations that postruminal supply of SH results in a disproportionate amount of the energy available for tissue gain being deposited as adipose (McLeod et al., 2001). It is our hypothesis that a substantial portion of
this increase in adipose tissue gain occurs in the visceral depots, and this is exacerbated by entry rate or simplicity of carbohydrate (i.e., monosaccharide vs.polysaccharide) delivered; this explains our rationale for inclusion of the postruminal glucose treatment.
Thus, for our experiment, a model was chosen that included feeding a basal diet, which allowed for growth and the maintenance of digestive tract integrity, supplemented with infused energy in the form of carbohydrate. This model was used to test 2 separate hypotheses. The first hypothesis is that digestive tract and liver mass are responsive to energy input with the extent of the response dependent upon site of carbohydrate infusion. The second hypothesis concerns our previous calorimetric observations that postruminal supply of SH results in a disproportionate amount of the energy available for tissue gain being deposited as adipose (McLeod et al., 2001). It is our hypothesis that a substantial portion ofthis increase in adipose tissue gain occurs in the visceral depots, and this is exacerbated by entry rate or simplicity of carbohydrate (i.e., monosaccharide vs.polysaccharide) delivered; this explains our rationale for inclusion of the postruminal glucose treatment.
การแปล กรุณารอสักครู่..
