In the mechanistic study of the effects of verbascoside on human
myelomonocytic leukaemia cells that have been exposed to proinflammatory
stimuli, such as LPS and interferon gamma, Speranza
et al. (2010) reported that its anti-inflammatory activities are dependent
on the inhibition of both the expression and activity of iNOS, the inhibition
of intracellular superoxide anion production and the suppression of
SOD, catalase, and glutathione peroxidase at the post-translational level.