The progenies used in the molecular analysis were those selected for CMD resistance based on phenotypic scoring and should be expected to express marker alleles or corresponding bands for the CMD2 gene. Rabbi et al. (2014) used a high density single
nucleotide polymorphism (SNP) map in a bi-parental mapping population segregating for the dominant monogenic resistance to CMD, and found a single locus with large effect, which is probably the CMD2 locus. In their study they found nearly half of the
progeny and the female parent to be resistant to CMD, with the remainder of the F1 individuals showing
disease symptoms ranging from mild to severe.