This American Thoracic society Statment was prepared by a committee of the scientific assembly on environmental and occupational health expert advisory group to emphasize the continuing importance of silicosis as a major lung disease worldwide and the need for increasing efforts in prevention. emphasis is placed on public health issues of silicosis rather than on importhat current research in pathologic mechanisms.
Diseases caused or contributed to by inhaiation of free crystalline sillica include sillicosis, pulmonary tuberculosis, industrial bronchitis with airflow limitation, and several extrapulmonary diseases. Chronic simple sillicosis, adiffuse fibronodular disease of the lung parenchyma, may be accompanied by subacute and chronic manifestations, including symptoms of dyspnea and cough; diffuse pulmonary nodulation symptoms of dyspnea and conglomerate lesions on chest roentgenograms and a ventilatory defect, usually opstructive, often with accompanying restrictive abriormality and reduced diffusing capacity. Acute and accelerated silicosis describe rapidly progressive forms, usually associated with intense silica exposure, These may be preceded or accompanied by silicoproteinosis, an alveolar filling process comparable to pulmonary alveolar proteinosis.
This American Thoracic society Statment was prepared by a committee of the scientific assembly on environmental and occupational health expert advisory group to emphasize the continuing importance of silicosis as a major lung disease worldwide and the need for increasing efforts in prevention. emphasis is placed on public health issues of silicosis rather than on importhat current research in pathologic mechanisms.Diseases caused or contributed to by inhaiation of free crystalline sillica include sillicosis, pulmonary tuberculosis, industrial bronchitis with airflow limitation, and several extrapulmonary diseases. Chronic simple sillicosis, adiffuse fibronodular disease of the lung parenchyma, may be accompanied by subacute and chronic manifestations, including symptoms of dyspnea and cough; diffuse pulmonary nodulation symptoms of dyspnea and conglomerate lesions on chest roentgenograms and a ventilatory defect, usually opstructive, often with accompanying restrictive abriormality and reduced diffusing capacity. Acute and accelerated silicosis describe rapidly progressive forms, usually associated with intense silica exposure, These may be preceded or accompanied by silicoproteinosis, an alveolar filling process comparable to pulmonary alveolar proteinosis.
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