Our data show that G1/0 cell cycle arrest occurred in the
control after 72 h, i.e., when the culture reached confluence
and there was no room for the cells to multiply. The irradiated
culture did not experience cell cycle arrest after 72 h because of
dramatically decreased viability and slower growth caused by
cell cycle arrest prevented the culture from filling the culture
dish. Our data on gH2AX showed clearly elevated levels, and
this is a certain sign of DSBs as well as of reparation efforts
undertaken by the cell [13]. Strongest presence of gH2AX
already at 1 h after irradiation showed that DSB were formed
immediately after the irradiation procedure. Those levels
decreased in time and reached the control level after 72 h,
when, as it can be assumed, all DSB repair procedures were
finished. Confirmation of the finished repair process was
provided by the released cell cycle after 72 h.