We demonstrated that PRMT5-mediated histone arginine methylation is required to elicit ZNF224 transcriptional repression. In this study, we show that KAP1 interacts with PRMT5 and is a novel substrate for PRMT5 methylation.
We demonstrated that PRMT5-mediated histone argininemethylation is required to elicit ZNF224 transcriptional repression. In this study, we showthat KAP1 interacts with PRMT5 and is a novel substrate for PRMT5 methylation.