) During the subsequent 24 to
48 hours (three to four day, after ingestion),
hepatocellular damage and renal impairment
progressively worsen, and jaundice, hypoglycemia,
oliguria, delirium and confusion develop. Myocardiopathy
and coagulopathy may be present, and
convulsions, coma and death may occur. The
mortality rate varies from 40 to 90 percent.,"1
Death from A. phalloides-type poisoning is usually
the result of hepatic or renal failure or
both. 1