(Fig. 1). At these concentrations, Hx reduced the
rate of bacterial growth because entry into the lag phase was
significantly (P < 0.05) delayed compared with the control and
0.06mMtreatments. Concentrations greater than 3mMcompletely
inhibited Xcc growth. Hx at 0.6, 1.5 and 3 mM apparently had a
temporary bacteriostatic effect which may explain the reduction in
the rate of bacterial growth. Assay with the Live/Dead Cell Viability
kit indicated that concentrations greater than 3 mM Hx did not kill
Xcc cells.