3.7. Flesh color, firmness, SSC and TA
Chromaticity L* in both control and NO-treated fruit gradually declined through 10 d storage. A slight inhibition in chromaticity L* decline was noted for NO-treated fruit, but treatment differences were statistically significant difference only from 6 to 8 d of storage (Fig. 5A). Chromaticity a* and b* values increased continuously in both control and NO-treated fruit, indicating change in flesh color from pale yellow to more intense yellow as fruit ripened. NO treatment, however, strongly suppressed flesh yellowing (Fig. 5B and C). Consistent with suppressed color change of NO-treated fruit during storage, changes in other ripening parameters including softening (Fig. 6A), SSC accumulation (Fig. 6B) and decline in TA (Fig. 6C) also reflected NO-mediated delay in mango ripening
3.7. Flesh color, firmness, SSC and TA Chromaticity L* in both control and NO-treated fruit gradually declined through 10 d storage. A slight inhibition in chromaticity L* decline was noted for NO-treated fruit, but treatment differences were statistically significant difference only from 6 to 8 d of storage (Fig. 5A). Chromaticity a* and b* values increased continuously in both control and NO-treated fruit, indicating change in flesh color from pale yellow to more intense yellow as fruit ripened. NO treatment, however, strongly suppressed flesh yellowing (Fig. 5B and C). Consistent with suppressed color change of NO-treated fruit during storage, changes in other ripening parameters including softening (Fig. 6A), SSC accumulation (Fig. 6B) and decline in TA (Fig. 6C) also reflected NO-mediated delay in mango ripening
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