Overproduction of ROS can damage important biomolecules,
such as DNA, proteins and lipids, and initiate a cascade of
events, bringing about impaired cellular function (Liu et al., 2014).
To counteract oxidative stress and keep cellular redox state in
balance, cells have evolved antioxidant defenses that act at different
levels to prevent or repair such damage. Under normal
physiological states, ROS is rapidly eliminated by antioxidant enzymes,
such as superoxide dismutase (SOD), catalase (CAT), glutathione
peroxidase (GPx) and glutathione reductase (GR)