It is possible that this was brought about by a modification of the inflammatory reaction or organization of the fibrin network in the tissue spaces at early stages of inflammatory phase of healing by the Av extract, which may act as a ‘scaffold’or template for fibroblast activity. It also appears that Av may have enhanced the return of
cellularity to within normal level. This could merely reflect the anti-inflammatory effect of the drug in reducing the number of adventitious phagocytic cells in the area.