After 14-d and 28-d exposure, the biomass and shell diameter
growth of A. fulica in the 40 mg kg1 and above treatments were
significantly reduced (p < 0.05; ANOVA; Figs. 1 and 2). After a 28-
d exposure, the total food intake in the 40 mg kg1 and above
treatments were significantly reduced (p < 0.05; ANOVA; Fig. 3).
Based on the statistical analysis, the NOECs of TCS for the biomass,
shell diameter growth, and total food intake were 24 mg kg1. The
NOEC of TCS for 28-d survival was 200 mg kg1. After a 14-d exposure,
there was no mortality in any treatment. Therefore, the 14-d
acute EC50 for survival of A. fulica was much larger than
340 mg kg1.
3.2. Biochemical
After 14-d and 28-d exposure, the biomass and shell diameter
growth of A. fulica in the 40 mg kg1 and above treatments were
significantly reduced (p < 0.05; ANOVA; Figs. 1 and 2). After a 28-
d exposure, the total food intake in the 40 mg kg1 and above
treatments were significantly reduced (p < 0.05; ANOVA; Fig. 3).
Based on the statistical analysis, the NOECs of TCS for the biomass,
shell diameter growth, and total food intake were 24 mg kg1. The
NOEC of TCS for 28-d survival was 200 mg kg1. After a 14-d exposure,
there was no mortality in any treatment. Therefore, the 14-d
acute EC50 for survival of A. fulica was much larger than
340 mg kg1.
3.2. Biochemical
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