Discussion : Pathophysiologically, OVT is explained by Virchow’s triad, because pregnancy is associated with a
hypercoagulable state, venous stasis due to compression of the inferior vena cava by the uterus and endothelial
trauma during delivery or from local inflammation. Common symptoms and signs of OVT include lower abdomen
or flank pain, fever and leukocytosis usually within the first ten days after delivery. The reported incidence of OVT
ranges 0,05-0,18% of pregnancies and in most cases the right ovarian vein is the one affected. Anticoagulation and
antibiotics is the mainstay of treatment of OVT. Complications of OVT include sepsis, extension of the thrombus to
the inferior vena cava and renal veins, and pulmonary embolism. The incidence of pulmonary embolism is
reported to be 13.2% and represents the main source of mortality due to OVT.
Discussion : Pathophysiologically, OVT is explained by Virchow’s triad, because pregnancy is associated with ahypercoagulable state, venous stasis due to compression of the inferior vena cava by the uterus and endothelialtrauma during delivery or from local inflammation. Common symptoms and signs of OVT include lower abdomenor flank pain, fever and leukocytosis usually within the first ten days after delivery. The reported incidence of OVTranges 0,05-0,18% of pregnancies and in most cases the right ovarian vein is the one affected. Anticoagulation andantibiotics is the mainstay of treatment of OVT. Complications of OVT include sepsis, extension of the thrombus tothe inferior vena cava and renal veins, and pulmonary embolism. The incidence of pulmonary embolism isreported to be 13.2% and represents the main source of mortality due to OVT.
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