(van den Heuvel and Waterreus, 1985). The first phase includes adhesion, germination, penetration and killing the host epidermal cells, which leads to primary lesion formation 24 h after inoculation. In the second phase, B. cinerea grows slowly without new primary lesion formation until 72 h. In the third phase, the primary lesion is able to grow out into the spreading lesion after 72 h. Our results suggest that P. terrestris extract exerted its inhibitory activity during the early phase of infection, resulting in inhibited spore germination.