ply Link me no,in my research DNB task, I got the Gamma activity in fr การแปล - ply Link me no,in my research DNB task, I got the Gamma activity in fr ไทย วิธีการพูด

ply Link me no,in my research DNB t

ply Link me
no,in my research DNB task, I got the Gamma activity in frontal lobe and parietal lobe . Frequency about 40-46 Hz.Theta was found in frontal lobe coupling with Gamma wave.
? | 5 years ago
Reply
I did some research and read some posts and WM training has shown to increase theta waves in the brain. I have an interesting quote here from a website, that explains why you may feel tired when you play N-Back.
"People with ADD/ADHD [inattentive ADD/ADHD, as opposed to other types of ADD such as overfocused] exhibit too little Beta (thinking) activity and too much Theta(dreaming) activity. This is the classic inattention for ADD. Too much theta compared to beta, which can cause attention problems and hyperactivity.

(It seems counterintuitive that too much slow wave can cause a child to be hyperactive, but because the brain is operating too slowly and not getting enough stimuli, the child seeks external stimulation to reward his brain.)

I've concluded from reading this, when you play n-back you are increase theta (a dream state of mind in your brain) and thats why you feel tired, and thats why you have more vivid dreaming when you go to sleep.
The thing that worries me is i have no attention problem, but I'm afraid if I constantly increase my theta waves in my brain I could obtain attention problems. What do you guys think? If you would like to read the whole article heres the link.
http://www.causeof.org/brainwaves.htm
87d0tc0m | 6 years ago
Reply
no,in my research the DNB task, I got the Gamma activity in frontal lobe and parietal lobe . Frequency about 40-46 Hz.Theta was found in frontal lobe coupling with Gamma wave.
? | 5 years ago
Reply
Normal activation of D2 receptors will not decrease D2-receptor density, it will increase it. I understand that a *sufficient* drug-induced increase in receptor activation may lead to smaller numbers of *active* receptor sites, when the receptors cannot desensitize sufficiently, but typical activation, as through mental force, should not exceed the increased threshold resultant by initial up-population/desensitization in response to greater neurotransmission (down-regulation)--only when activity is much too high, as in drug overdose, will neurons react by -shutting down- receptor sites (receptor downgrading), to compensate for the excess activity.
D2 receptor sites provide frontal lobe inhibition involved in tasks such as insight into one's condition, the multiply-coordinated effort in the frontal, executive brain--when the frontal lobe is inhibited, information from other brain regions can be adapted. They also play an excitatory role in the limbic system, in the more primitive lower part of the brain.
Practice results in lower levels of brain activity during task execution, a result of the incorporation of larger populations of less sensitive receptors. Larger populations do not all exhibit electrical activity at once; if they did, as in a drug-induced scenario, it may result in downgrading of receptor sites to normalize activity levels.
D1 and D2 receptors are contrary in their effect on electrical activity though only in the frontal brain, but activity between them is not directly mediated; an increase in D2 activity is more likely to not have an effect on D1 transmission, and vice-versa.

Also, ADD, with all its paradoxes, is the result of an underlying disorder, and increased theta, if it is the result of WM training, would not have the same dysfunctional profile--as it is "opportunistically" integrated into the normal brain.

N.B. An increase in receptor population normally coincides with down-regulation: up-population and desensitization.

Too much theta vs. beta does not *cause* inattention, etc., it *coincides* with it in some cases.

WT
? | 6 years ago
Reply
Receptor population increases initially with neurotransmitter levels and decreases again when activity is too high. I remember this being counter-intuitive (-in class).

Receptors become downgraded when they are damaged by excessive activity. This is an additional effect--damaged receptors.

N.B. I learned this instantly (from class) and never checked (I read textbooks). I'm having trouble recalling the professor's specific association from class. This was 'do expert.' I let neuroscience go.

Sorry for any 'garbåge,' Good Luck!!

Have to remember why...

WT

OK now I believe this is right... D2-? Insight into one's condition-? That's inference.

TY /gap junctions...
? | 6 years ago
Reply
Dude, you really sit down in the Couch of Context.

It has been shown by Klingberg et al that D1 receptor density binding merely modulates with training (that is, it either increases or decreases depending on the person).

http://www.klingberglab.se/pub/McNab2009.pdf

Get a hold of yourself, man!

The brain isn't at all counterintuitive; it is only so when you haven't understood some of the basic organizational features thereof. Let the prejudices go.
? | 6 years ago
Reply
I meant I am not certain, it's been a long time since I first encountered this material--

...I don't think the results showed modulation per se, there was a negative correlation between WM training and D1 BP... This should be unrelated to receptor density, though I have read that cortical D1 receptor site density was found to increase with WM training. It could be two effects, or I could be mistaken--receptor density binding does not exist...

...What was explained to be counter-intuitive is that receptor population actually decreases when activity is too high. "Downstream" effect-.

...I doubt this was emotional; Making sure the logic was correct (valid)...
I also explained that I was taught this:
-Thx-
? | 6 years ago
Reply
It isn't counterintuitive to me that overactivation leads to reduced receptor populations. Homoeostasis is a very well understood biological phenomenon.

It isn't that your posts were emotional either. They are poorly composed and do not convey whatever points you're trying to make very clearly.

Use more active verbs. Get out of the linguistic Dark Age your schooling has given you.
? | 6 years ago
Reply
I'm certainly no neuroscientist, but notwithstanding gap junctions/electrical synapses, communication between neurons is mediated by ionotropic and metabotropic receptors, which in the absence or dearth of their endogenous ligands, the brain being replete with homeostatic mechanisms as it is, upregulate - which is to say they become more sensitive to their respective ligands. To me, increased theta activity implies increase D2 receptor activation within the prefrontal cortex. D1 receptors and D2 receptors are in a real sense opposites of one another: the former, in binding to heterotrimeric g proteins with an alpha-s domain, increases intracellular cAMP concentrations. The latter by binding to g-proteins with an inhibitory type alpha domain, decrease cAMP leves. The former promotes release of acetylcholine and generally increases glutamatergia. The latter presumably decrease the release of acetylcholine and dampens glutamatergic currents (which is where the theta waves come in). There is, for reasons a bit to complicated to explain here, an inverse relationship between the density of these receptors. To the extent to which you activate D2 receptors, you decrease D2 receptor density and increase D1 receptor density. As I said, theta waves, to me, imply D2 receptor activation and with sufficient stimulation of neurons through these receptors, you can expect to find a downregulation of D2 receptors and an upregulation of D1. It just so happens that research into dual-n-back tests has shown that it results in an upregulation of D1 receptors.

In other words, your theory that the preponderance of theta wave states is proportional to the amount of time already spent in theta states appears - at least to me, who am a dilettante and not much better than a layman, at that, in these matters - to be diametric opposition to reality. The more time one spends doing n-back tests - i.e. being in a theta state - the less likely one is to be in such a state when not taking them.
? | 6 years ago
Reply
Lots of typos and poor compositional choices. My apologies. I was in a bit of a hurry.
? | 6 years ago
Reply
http://www.klingberglab.se/pub/McNab2009.pdf

You would do well to reference published material...
? | 6 years ago
Reply
You shouldn't worry. Theta activity is a waveform associated with concentration:

http://www.mindupdate.com/?p=133

Relatedly, I find that at times when I listen to binaural beats at theta, I can concentrate on the material slightly more easily.
? | 6 years ago
Reply
"...the material I'm reading..."
? | 6 years ago
Reply
Athletes rest at lower frequency than do sedentary persons. The brain must become bored,--also due to fitness.

Highly creative people are often bored, meditative persons are--ADD--, etc....truck drivers...


Theta is a basal openness: to possibilities otherwise present.

--N-back theta should folloW Training...
? | 6 years ago
Reply
Wrong, wrong, wrong. There is no boredom in the associative process. It is a natural outcome of a relaxed state of mind.
? | 6 years ago
Reply
Relaxation is also a qualified state; the fundamental divergence of the normal state in trained individuals can be attributed to contextual boredom-; as the comparative state, boredom is proficient, relaxation--deniably-so. The operative conclusion is that "descriptive" activity should not be consubstantiated into further activity suggesting the culpable successive state._
**
The primary description initiated is accounted to boredom. Relaxation indicates a primary participatory relevance by its necessary qualification. The proper correlate of relaxation in this case is boredom--activity significantly-is absent (participatory-.):
...[^]
-WT
? | 6 years ago
Reply
Nop
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ply Link me no,in my research DNB task, I got the Gamma activity in frontal lobe and parietal lobe . Frequency about 40-46 Hz.Theta was found in frontal lobe coupling with Gamma wave. ? | 5 years ago Reply I did some research and read some posts and WM training has shown to increase theta waves in the brain. I have an interesting quote here from a website, that explains why you may feel tired when you play N-Back. "People with ADD/ADHD [inattentive ADD/ADHD, as opposed to other types of ADD such as overfocused] exhibit too little Beta (thinking) activity and too much Theta(dreaming) activity. This is the classic inattention for ADD. Too much theta compared to beta, which can cause attention problems and hyperactivity.(It seems counterintuitive that too much slow wave can cause a child to be hyperactive, but because the brain is operating too slowly and not getting enough stimuli, the child seeks external stimulation to reward his brain.)I've concluded from reading this, when you play n-back you are increase theta (a dream state of mind in your brain) and thats why you feel tired, and thats why you have more vivid dreaming when you go to sleep. The thing that worries me is i have no attention problem, but I'm afraid if I constantly increase my theta waves in my brain I could obtain attention problems. What do you guys think? If you would like to read the whole article heres the link. http://www.causeof.org/brainwaves.htm 87d0tc0m | 6 years ago Reply no,in my research the DNB task, I got the Gamma activity in frontal lobe and parietal lobe . Frequency about 40-46 Hz.Theta was found in frontal lobe coupling with Gamma wave. ? | 5 years ago Reply Normal activation of D2 receptors will not decrease D2-receptor density, it will increase it. I understand that a *sufficient* drug-induced increase in receptor activation may lead to smaller numbers of *active* receptor sites, when the receptors cannot desensitize sufficiently, but typical activation, as through mental force, should not exceed the increased threshold resultant by initial up-population/desensitization in response to greater neurotransmission (down-regulation)--only when activity is much too high, as in drug overdose, will neurons react by -shutting down- receptor sites (receptor downgrading), to compensate for the excess activity. D2 receptor sites provide frontal lobe inhibition involved in tasks such as insight into one's condition, the multiply-coordinated effort in the frontal, executive brain--when the frontal lobe is inhibited, information from other brain regions can be adapted. They also play an excitatory role in the limbic system, in the more primitive lower part of the brain. Practice results in lower levels of brain activity during task execution, a result of the incorporation of larger populations of less sensitive receptors. Larger populations do not all exhibit electrical activity at once; if they did, as in a drug-induced scenario, it may result in downgrading of receptor sites to normalize activity levels.D1 and D2 receptors are contrary in their effect on electrical activity though only in the frontal brain, but activity between them is not directly mediated; an increase in D2 activity is more likely to not have an effect on D1 transmission, and vice-versa.Also, ADD, with all its paradoxes, is the result of an underlying disorder, and increased theta, if it is the result of WM training, would not have the same dysfunctional profile--as it is "opportunistically" integrated into the normal brain.N.B. An increase in receptor population normally coincides with down-regulation: up-population and desensitization.Too much theta vs. beta does not *cause* inattention, etc., it *coincides* with it in some cases.WT ? | 6 years ago Reply Receptor population increases initially with neurotransmitter levels and decreases again when activity is too high. I remember this being counter-intuitive (-in class). Receptors become downgraded when they are damaged by excessive activity. This is an additional effect--damaged receptors.N.B. I learned this instantly (from class) and never checked (I read textbooks). I'm having trouble recalling the professor's specific association from class. This was 'do expert.' I let neuroscience go.
Sorry for any 'garbåge,' Good Luck!!

Have to remember why...

WT

OK now I believe this is right... D2-? Insight into one's condition-? That's inference.

TY /gap junctions...
? | 6 years ago
Reply
Dude, you really sit down in the Couch of Context.

It has been shown by Klingberg et al that D1 receptor density binding merely modulates with training (that is, it either increases or decreases depending on the person).

http://www.klingberglab.se/pub/McNab2009.pdf

Get a hold of yourself, man!

The brain isn't at all counterintuitive; it is only so when you haven't understood some of the basic organizational features thereof. Let the prejudices go.
? | 6 years ago
Reply
I meant I am not certain, it's been a long time since I first encountered this material--

...I don't think the results showed modulation per se, there was a negative correlation between WM training and D1 BP... This should be unrelated to receptor density, though I have read that cortical D1 receptor site density was found to increase with WM training. It could be two effects, or I could be mistaken--receptor density binding does not exist...

...What was explained to be counter-intuitive is that receptor population actually decreases when activity is too high. "Downstream" effect-.

...I doubt this was emotional; Making sure the logic was correct (valid)...
I also explained that I was taught this:
-Thx-
? | 6 years ago
Reply
It isn't counterintuitive to me that overactivation leads to reduced receptor populations. Homoeostasis is a very well understood biological phenomenon.

It isn't that your posts were emotional either. They are poorly composed and do not convey whatever points you're trying to make very clearly.

Use more active verbs. Get out of the linguistic Dark Age your schooling has given you.
? | 6 years ago
Reply
I'm certainly no neuroscientist, but notwithstanding gap junctions/electrical synapses, communication between neurons is mediated by ionotropic and metabotropic receptors, which in the absence or dearth of their endogenous ligands, the brain being replete with homeostatic mechanisms as it is, upregulate - which is to say they become more sensitive to their respective ligands. To me, increased theta activity implies increase D2 receptor activation within the prefrontal cortex. D1 receptors and D2 receptors are in a real sense opposites of one another: the former, in binding to heterotrimeric g proteins with an alpha-s domain, increases intracellular cAMP concentrations. The latter by binding to g-proteins with an inhibitory type alpha domain, decrease cAMP leves. The former promotes release of acetylcholine and generally increases glutamatergia. The latter presumably decrease the release of acetylcholine and dampens glutamatergic currents (which is where the theta waves come in). There is, for reasons a bit to complicated to explain here, an inverse relationship between the density of these receptors. To the extent to which you activate D2 receptors, you decrease D2 receptor density and increase D1 receptor density. As I said, theta waves, to me, imply D2 receptor activation and with sufficient stimulation of neurons through these receptors, you can expect to find a downregulation of D2 receptors and an upregulation of D1. It just so happens that research into dual-n-back tests has shown that it results in an upregulation of D1 receptors.

In other words, your theory that the preponderance of theta wave states is proportional to the amount of time already spent in theta states appears - at least to me, who am a dilettante and not much better than a layman, at that, in these matters - to be diametric opposition to reality. The more time one spends doing n-back tests - i.e. being in a theta state - the less likely one is to be in such a state when not taking them.
? | 6 years ago
Reply
Lots of typos and poor compositional choices. My apologies. I was in a bit of a hurry.
? | 6 years ago
Reply
http://www.klingberglab.se/pub/McNab2009.pdf

You would do well to reference published material...
? | 6 years ago
Reply
You shouldn't worry. Theta activity is a waveform associated with concentration:

http://www.mindupdate.com/?p=133

Relatedly, I find that at times when I listen to binaural beats at theta, I can concentrate on the material slightly more easily.
? | 6 years ago
Reply
"...the material I'm reading..."
? | 6 years ago
Reply
Athletes rest at lower frequency than do sedentary persons. The brain must become bored,--also due to fitness.

Highly creative people are often bored, meditative persons are--ADD--, etc....truck drivers...


Theta is a basal openness: to possibilities otherwise present.

--N-back theta should folloW Training...
? | 6 years ago
Reply
Wrong, wrong, wrong. There is no boredom in the associative process. It is a natural outcome of a relaxed state of mind.
? | 6 years ago
Reply
Relaxation is also a qualified state; the fundamental divergence of the normal state in trained individuals can be attributed to contextual boredom-; as the comparative state, boredom is proficient, relaxation--deniably-so. The operative conclusion is that "descriptive" activity should not be consubstantiated into further activity suggesting the culpable successive state._
**
The primary description initiated is accounted to boredom. Relaxation indicates a primary participatory relevance by its necessary qualification. The proper correlate of relaxation in this case is boredom--activity significantly-is absent (participatory-.):
...[^]
-WT
? | 6 years ago
Reply
Nop
การแปล กรุณารอสักครู่..
ผลลัพธ์ (ไทย) 3:[สำเนา]
คัดลอก!
ชั้นเชื่อมโยงชั้น
ไม่ ในงานวิจัยของฉันดีเอ็นบี งานผมมีแกมมากิจกรรมในสมองส่วนหน้า และสมองกลีบข้าง . ความถี่ประมาณ 40-46 เฮิร์ต ทีพบในคู่ด้านหน้ากับคลื่นแกมมา
? | เมื่อ 5 ปีที่แล้ว

ตอบฉันได้บางวิจัยและอ่านบางกระทู้และ WM ฝึกได้แสดงการเพิ่มคลื่น theta ในสมอง ผมมีอ้างอิงที่น่าสนใจจากเว็บไซต์นั่นอธิบายว่าทำไมคุณอาจจะรู้สึกเหนื่อยเมื่อคุณเล่น n-back .
" คนเพิ่ม / ADHD [ เพิกเฉย ADD / ADHD , เมื่อเทียบกับประเภทอื่น ๆ เพิ่ม เช่น overfocused ] แสดงน้อยเกินไปเบต้า ( คิด ) กิจกรรมมากเกินไปและ Theta ( ฝัน ) กิจกรรม นี่คือการปล่อยปละละเลยคลาสสิกเพื่อเพิ่ม มากเกินไป theta เมื่อเทียบกับเบต้า ซึ่งอาจก่อให้เกิดปัญหาความสนใจและสมาธิสั้น .

( ดูเหมือน counterintuitive ที่คลื่นช้ามากเกินไปจะทำให้เด็กเป็นไฮเปอร์ แต่เพราะสมองเป็นปฏิบัติการช้าเกินไปและไม่ได้รับการกระตุ้น พอเด็กพยายามกระตุ้นจากภายนอกเพื่อให้รางวัลแก่สมอง ของเขา )

ผมสรุปจากการอ่านนี้ เมื่อคุณเล่น n-back คุณเพิ่มขึ้น Theta ( ความฝันของรัฐ ความคิดในสมองของคุณและทำให้คุณรู้สึกเหนื่อยและ thats เหตุผลที่คุณจะได้สดใสขึ้น ฝัน เมื่อคุณจะเข้านอน
สิ่งที่ผมกังวล คือ ผมมีปัญหาไม่มีความสนใจ แต่ฉันกลัวว่าถ้าฉันตลอดเวลาเพิ่มของฉันคลื่น theta ในสมองของฉัน ฉันอาจได้รับปัญหาที่สนใจ พวกคุณคิดว่ายังไง ถ้าคุณต้องการอ่านบทความทั้งหมดนี่คือการเชื่อมโยง

87d0tc0m http://www.causeof.org/brainwaves.htm | 6 ปีที่แล้ว

ตอบไม่ในการวิจัยของฉันดีเอ็นบีงาน ผมมีแกมมากิจกรรมในสมองส่วนหน้า และสมองกลีบข้าง . ความถี่ประมาณ 40-46 เฮิร์ต ทีพบในคู่ด้านหน้ากับคลื่นแกมมา
? | 5 ปีที่แล้ว

ปกติตอบการกระตุ้นตัวรับ D2 จะไม่ลดความหนาแน่นของตัวรับ D2 ก็จะเพิ่มขึ้นเองฉันเข้าใจว่า * เพียงพอ * ยา - เทพเพิ่มตัวรับการกระตุ้นอาจนำไปสู่ตัวเลขขนาดเล็กของเว็บไซต์ที่ใช้งาน * * รับ เมื่อผู้รับไม่สามารถเป็นหนึ่งได้พอสมควร แต่โดยทั่วไปการเปิดใช้งาน ผ่านจิตบังคับ
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