metabolic acidosis (pH:7.09) with an elevated anion gap
(27.8 mmol/L) and an increased base excess (-25.2 mmol/L).
Results of biochemical analyses were within normal limits
except for hyperglycemia and mildly increased levels of urea,
creatinine and uric acid. Ketonuria was also present (Table 1).
Based on typical laboratory findings such as hyperglycemia,
ketonuria, ketonemia, metabolic acidosis and clinical features
such as vomiting and encephalopathy, a diagnosis of DKA was
considered and treatment with intravenous fluids and insulin
was started