Maternal P2 backfat depth (Figure 2; overall mean: 15.3 ± 0.1 mm) at d 25 of pregnancy, before commencement of injections, did not differ between parities (P > 0.8) or treatment groups (P > 0.9), but varied between replicate weeks (P ≤ 0.001), and effects of parity also varied between replicate weeks (P = 0.001). Maternal P2 backfat depth at d 50 of pregnancy was also unaffected by parity or treatment (P > 0.4 for each) and varied with replicate week (P ≤ 0.001), and effects of parity varied with replicate week (P ≤ 0.001). Dams treated with pST from d 25 to 100 of pregnancy gained P2 backfat depth more slowly between d 50 and 109 of pregnancy than did control dams (P = 0.015), whereas the rate of P2 backfat gain from d 25 to 109 was not different from controls in dams whose pST treatment stopped at d 50 (P > 0.6). Consequently, maternal P2 backfat depth before farrowing was greater in gilts than sows (P = 0.001) and was reduced in dams treated with pST from d 25 to 100 of pregnancy (P = 0.001), but was not reduced in those treated from d 25 to 50 only (P = 0.165) and varied with replicate week (P ≤ 0.001). Rate of maternal P2 backfat loss during lactation did not differ between treatments (P > 0.6) or parities (P > 0.2), but did vary between replicate weeks (P ≤ 0.001). Consequently, maternal P2 backfat depth at weaning (Figure 2) was less in dams treated with pST from d 25 to 100 than in controls (P = 0.034), but was not reduced in dams whose treatment was finished at d 50 (P > 0.6).
Maternal P2 backfat depth (Figure 2; overall mean: 15.3 ± 0.1 mm) at d 25 of pregnancy, before commencement of injections, did not differ between parities (P > 0.8) or treatment groups (P > 0.9), but varied between replicate weeks (P ≤ 0.001), and effects of parity also varied between replicate weeks (P = 0.001). Maternal P2 backfat depth at d 50 of pregnancy was also unaffected by parity or treatment (P > 0.4 for each) and varied with replicate week (P ≤ 0.001), and effects of parity varied with replicate week (P ≤ 0.001). Dams treated with pST from d 25 to 100 of pregnancy gained P2 backfat depth more slowly between d 50 and 109 of pregnancy than did control dams (P = 0.015), whereas the rate of P2 backfat gain from d 25 to 109 was not different from controls in dams whose pST treatment stopped at d 50 (P > 0.6). Consequently, maternal P2 backfat depth before farrowing was greater in gilts than sows (P = 0.001) and was reduced in dams treated with pST from d 25 to 100 of pregnancy (P = 0.001), but was not reduced in those treated from d 25 to 50 only (P = 0.165) and varied with replicate week (P ≤ 0.001). Rate of maternal P2 backfat loss during lactation did not differ between treatments (P > 0.6) or parities (P > 0.2), but did vary between replicate weeks (P ≤ 0.001). Consequently, maternal P2 backfat depth at weaning (Figure 2) was less in dams treated with pST from d 25 to 100 than in controls (P = 0.034), but was not reduced in dams whose treatment was finished at d 50 (P > 0.6).
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