To date, researchers have failed to perform hemodynamic investigations within the first 60 minutes of an embolic event in humans; hence the observations made in animals cannot be confronted with (potential) clinical findings. The experimental model demonstrates the predominance of right ventricular failure [13]; however, this may differ in humans. Clark [14] has assumed that hemodynamic disturbances in human subjects have a two-phase course: (a) initially, symptoms of right ventricular failure are observed (pulmonary artery hypertension, increased blood pressure in large veins); (b) later, left ventricular failure develops, especially left atrial failure with pulmonary hypertension which is referred to as secondary hypertension (due to left atrial or/and ventricular failure). Approximately 10 years