This study was conducted to investigate the effect of dietary vitamin E concentration on growth performance, iron-catalyzed lipid
peroxidation in liver and muscle tissue, and erythrocyte fragility of transgenic growth hormone coho salmon (Oncorhynchus kisutch). Fish
were fed one of four isoenergetic and isonitrogenous experimental diets that contained either 11, 29, 50, or 105 IU of vitamin E/kg. Following
the 10-week feeding trial, no significant ( PN0.05) diet-related differences were detected in growth, whole body proximate composition or
erythrocyte fragility. The vitamin E contents of liver and muscle, however, were affected by the dietary treatment. Fish fed diets containing
z50 IU of vitamin E/kg had significantly increased vitamin E concentrations in their tissues. Iron-catalyzed lipid peroxidation of liver and
muscle tissue of fish fed elevated dietary vitamin E (z50 IU vitamin E/kg diet) was significantly lower ( Pb0.05) than that noted for fish fed
the diet containing no supplemental vitamin E. The results indicated that changes in tissue lipid peroxidation measurements precede clinical
signs of sub-optimal vitamin E intake.
This study was conducted to investigate the effect of dietary vitamin E concentration on growth performance, iron-catalyzed lipid
peroxidation in liver and muscle tissue, and erythrocyte fragility of transgenic growth hormone coho salmon (Oncorhynchus kisutch). Fish
were fed one of four isoenergetic and isonitrogenous experimental diets that contained either 11, 29, 50, or 105 IU of vitamin E/kg. Following
the 10-week feeding trial, no significant ( PN0.05) diet-related differences were detected in growth, whole body proximate composition or
erythrocyte fragility. The vitamin E contents of liver and muscle, however, were affected by the dietary treatment. Fish fed diets containing
z50 IU of vitamin E/kg had significantly increased vitamin E concentrations in their tissues. Iron-catalyzed lipid peroxidation of liver and
muscle tissue of fish fed elevated dietary vitamin E (z50 IU vitamin E/kg diet) was significantly lower ( Pb0.05) than that noted for fish fed
the diet containing no supplemental vitamin E. The results indicated that changes in tissue lipid peroxidation measurements precede clinical
signs of sub-optimal vitamin E intake.
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