Residual neuromuscular blockade is one of the
main causes of postoperative pulmonary and respiratory
complications, hypoxia, upper airway obstruction
and decreased oxygen saturation, which can increase
the incidence of tracheal re-intubation in critical care
units [4–9]. Sugammadex reverses neuromuscular
blockade more rapidly and reliably than acetylcholinesterase
inhibitors [3]. Anticholinergics are often
administered with neostigmine to counteract its muscarinic
side-effects, but in turn these may cause nausea
and vomiting, increased secretions, heart rhythm
abnormalities and bronchospasm.