The Dahlin et al. (2008b) findings are consistent with other fMRI research indicating that WM functioning in general as well as WM improvement is linked to the striatum. Pharmacological fMRI research is illustrative. Not surprisingly, this work involves challenges with DA compounds; the dopaminergic innervation from midbrain to striatum is particularly dense. Cools et al.(2007) showed that administration of bromocriptine, a DA D2 agonist, improved the flexible updating of WM representations and increased striatal BOLD activity. Conversely, Dodds et al. (2009) demonstrated that sulpiride, a DA antagonist, depressed striatal BOLD activity and affected WM performance. Murty et al. (2011) obtained direct evidence for a specific role of the striatum in updating as opposed to other WM processes. Using a new WM task,these investigators found selectively increased striatal BOLD activity during updating compared to maintenance and rewriting of information in WM. In general, these patterns are consistent with neurocomputational work suggesting that striatal neurons serve a key gating function in letting new information enter WM; the gat ecloses during memory maintenance and opens when the content of WM is to be updated (O’Reilly, 2006).