observed that mutations in Salmonella enterica fimbrial subunits genes led to reduced intestinal colonisation in chickens. Additionally, work by van der Velden et al. [17] showed that multiple fimbrial adhesions are required for a fully virulent profile in a murine model of Salmonella typhimurium infection. This attenuated cellular adhesion profile was confirmed for the C. pseudotuberculosis CP09 strain, and other experiments were conducted with the objective of determining the immunogenicity and protective potential of this mutant in a caseous lymphadenitis model in mice.