Hypertension developing in the second half of pregnancy is subdivided according to the presence or absence of co-existing significant proteinuria into PE and gestational hypertension (GH). Recent evidence suggests that PE can be further subdivided into early-PE and late-PE with the former being associated with a higher incidence of impaired placentation,6, 7, 8 fetal growth restriction9 and both short-term and long-term maternal mortality and morbidity.10, 11, 12