of COX-2 genes, which is directly related to the intensity
of organ inflammation (Poljakovic et al., 2001; Jana
et al., 2009), providing further evidence of increased gut
mucosa inflammation caused by E. coli. Therefore, the
present results support the clinical responses of E. coliinfected
pigs in our previous study, indicated by increased
white blood cells and serum proinflammatory cytokines
and acute phase proteins