Alzheimer’s disease (AD) is a progressive and complex neurodegenerative disorder characterized by the occurrence of senile plaques and neurofibrillary tangles. Amyloid beta (Ab) peptide is the
main component of senile plaques and is highly involved in the progression of AD. Accumulated evidences suggest that Abpeptide, in
the presence of biometals and oxygen, generates reactive oxygen
species and causes lipid peroxidation in neuronal cell membranes
(Butterfield and Lauderback, 2002; Rauk, 2008). Nowadays, the most
efficient therapeutic approach to AD is based on cholinesterase