The model of Affonce and Lutchen [39] is a static model focus- sing on asthma, AHR and airway hyper-sensitivity, and airway constriction. It is an extension of the work of Lambert et al. [16] taking account of the behaviour of not only the airway wall, but also increased parenchymal tethering due to constriction, elastic lung recoil, and ASM stress. While each of these elements are included in fairly simple and largely empirical ways, the assembled model has very important behaviour in terms of bistability in the