formed more compact structures than those formed by NP-LRS3. This observation could be attributed to the leaching of amylose, the loss of the amylopectin crystalline region during autoclaving,and re-association of the starch chains within the granules. Therefore,the expansion of starch granules to form tight structures within amorphous regions might reduce the susceptibility of LRS to enzymatic digestion. The gully shapes observed on the surfaces of GP-LRS3 and ZP-LRS3 might have been left by the hydrolysis of amylose on the surface of the weak tissue structure. HAMS has an irregular shape, and the sizes and surfaces of HAMS structures
resembled those of the structures formed by native starch. Notwithstanding the high amylose content of HAMS, given the absence of a re-crystallization process, its structure differed
substantially from that of resistant starch