Cysticercosis, infection with the larval stage of the pork tapeworm, Taenia solium, is a known cause of illness and death in humans (1,2). Neurocysticercosis, the most serious form of the disease, occurs when larvae invade the central nervous system. The disease is dependent on a 2-host life cycle in which humans serve as the definitive host and pigs as the intermediate host. Eggs, which are directly infectious, are shed in the feces of humans infected with the adult tapeworm. When these eggs are ingested by pigs, larvae emerge from the eggs, penetrate the intestinal mucosa, and disseminate through the bloodstream to various tissues where the larval stage, or cysticercus, develops. The cycle is completed when humans, the only naturally infected definitive host, consume raw or undercooked pork containing cysticerci, which attach to the small intestine and develop into the adult tapeworm. However, humans may also become infected with the larval stage when they ingest T. solium tapeworm eggs, typically in contaminated food or water. Cysticercosis is therefore a fecal–oral-transmitted disease acquired by ingestion of eggs excreted in the feces of a human tapeworm carrier