Other maternal causes include uterine factor (e.g., fibroid,
müllerian anomalies) [46, 47], maternal periodontal disease
[48], and genetic conditions, such as angiotensinogen gene
mutations. Prior history of SGA infant increases the risk of
recurrent SGA to up to 25 %. The women who were SGA at
birth have twofold increase for risk of fetal growth restriction
in their offspring [49].
Other maternal causes include uterine factor (e.g., fibroid,müllerian anomalies) [46, 47], maternal periodontal disease[48], and genetic conditions, such as angiotensinogen genemutations. Prior history of SGA infant increases the risk ofrecurrent SGA to up to 25 %. The women who were SGA atbirth have twofold increase for risk of fetal growth restrictionin their offspring [49].
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