It’s estimated that in the next few years one in 150 adults could have congenital heart disease (CHD).And two-thirds of those who have CHD are adults , according to a recent Canadian study. It is now know that problems can occur years after CHD repair and that ongoing cardiology follow-up can prevent or detect serious complications. But many patients whose CHD was repaired in childhood have not had regular cardiology follow-up. Indeed, many patients with CHD or with complications of CHD repair come to medical attention only by way of noncardiac care settings.
To provide high-quality care for these patients, in cardiac and noncardiac settings alike, nurses need to understand that long-term consequences can be relate either to the CHD itself or to the specific repair done.
This two-part article will review the anatomy, physiology, and approach to the repair of 10 common CHD lesions (also known as defects or anomalies : bicuspid aortic valve , atrial septal defect,ventricular septal defect, atrioventricular septal defect,coarctation of the aorta, pulmonic stenosis, tetralogy of fallot,transposition of the great arteries,congenitally corrected transposition of the great arteries, and single ventricle defect. Long-term outcomes of the treatments will be reviewed, identifying implications for nursing care. Part 1 provides an overview of six of the 10 conditions. For reference, Figure 1 depicts the normal heart
It’s estimated that in the next few years one in 150 adults could have congenital heart disease (CHD).And two-thirds of those who have CHD are adults , according to a recent Canadian study. It is now know that problems can occur years after CHD repair and that ongoing cardiology follow-up can prevent or detect serious complications. But many patients whose CHD was repaired in childhood have not had regular cardiology follow-up. Indeed, many patients with CHD or with complications of CHD repair come to medical attention only by way of noncardiac care settings.To provide high-quality care for these patients, in cardiac and noncardiac settings alike, nurses need to understand that long-term consequences can be relate either to the CHD itself or to the specific repair done.This two-part article will review the anatomy, physiology, and approach to the repair of 10 common CHD lesions (also known as defects or anomalies : bicuspid aortic valve , atrial septal defect,ventricular septal defect, atrioventricular septal defect,coarctation of the aorta, pulmonic stenosis, tetralogy of fallot,transposition of the great arteries,congenitally corrected transposition of the great arteries, and single ventricle defect. Long-term outcomes of the treatments will be reviewed, identifying implications for nursing care. Part 1 provides an overview of six of the 10 conditions. For reference, Figure 1 depicts the normal heart
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