A recent report demonstrated
that miR-196b expression is significantly more up-regulated in
AML than ALL and that, again, miR-196b up-regulation is negatively
associated with overall survival of AML patients [57]. In
addition to the miR-196b deregulation observed in leukaemia,
miR-196b has been reported to induce changes in myeloid development,
and the co-expression of miR-196b and miR-21
reportedly block granulocyte colony-stimulating factor-induced
granulopoiesis completely