In summary, we have demonstrated that the anti-inflammatory
mechanisms of Cinnamomum camphora may be able to
modulate macrophage-mediated inflammatory functions such
as the over-production of cytokines, NO and PGE2, adhesion
molecule activation and oxidative responses. Moreover, the
EtOAc fraction is the most promising one to isolate the active
anti-inflammatory principles. Finally, as antioxidant activity is
commonly related with gastroprotective (El-Abhar et al., 2003),
cytoprotective (Potapovich and Kostyuk, 2003) and hepatoprotective
effects (Sohn et al., 2003), the fractions or isolated compounds
from Cinnamomum camphora are worthy of additional
studies regard to these potential effects, under proper in vivo
models.