The infection of various plants with B. cinerea induces multiple
defense responses, which are mainly associated with
phytohormone-mediated defense pathways. In general, JA- and ETmediated
responses are involved in resistance against necrotrophic
pathogens, including B. cinerea [29,30]. However, several studies
have demonstrated that SA-signaling also plays a role in disease
resistance against B. cinerea [31,32]. Pretreatment of tomato plants
with ET or SA decreases the percentage of expanding lesions of the
pathogen, and increases transcription levels of CHI9 or CHI3,
respectively [33]. In the present study, we show that tomato plants