Apart from reducing ROS production, overexpression of SOD alsomitigated the activation of the JNK/AP1 pathway which has been implicated in ROS-induced mitochondrial injury and apoptotic cell death.
Apart from reducing ROS production, overexpression of SOD alsomitigated the activation of the JNK/AP1 pathway which has been implicated in ROS-induced mitochondrial injury and apoptotic celldeath.