Two experiments were carried out with
broiler breeders (experiment 1) and laying hens (experiment
2) to study the effects of Se sources, in interaction
with dietary level of Se or dietary fats on performance,
Se incorporation into tissues (blood, liver, breast muscle,
and egg) and eggs, hatchability, and glutathione
peroxidase (GPX) activities in tissues and blood. Both
experiments involved a 3 × 2 factorial arrangement of 3
Se sources (selenite, Se yeast, or B-Traxim Se) and either
2 levels of each source (0.1 or 0.3 mg/kg) or 2 fats
(fresh or oxidized). Egg production was not affected
by Se source or dietary fat in both experiments. Egg
production was greater (P < 0.01) in breeder hens fed
0.3 mg/kg of Se in experiment 1. Hatchability of eggs
from hens fed 0.1 mg/kg of Se was lower (P < 0.05) in
hens fed Se yeast, whereas from hens fed 0.3 mg/kg of
Se, it was comparable across treatments. Selenium in
egg, liver, and breast muscle was greater (P < 0.01 or
Two experiments were carried out with
broiler breeders (experiment 1) and laying hens (experiment
2) to study the effects of Se sources, in interaction
with dietary level of Se or dietary fats on performance,
Se incorporation into tissues (blood, liver, breast muscle,
and egg) and eggs, hatchability, and glutathione
peroxidase (GPX) activities in tissues and blood. Both
experiments involved a 3 × 2 factorial arrangement of 3
Se sources (selenite, Se yeast, or B-Traxim Se) and either
2 levels of each source (0.1 or 0.3 mg/kg) or 2 fats
(fresh or oxidized). Egg production was not affected
by Se source or dietary fat in both experiments. Egg
production was greater (P < 0.01) in breeder hens fed
0.3 mg/kg of Se in experiment 1. Hatchability of eggs
from hens fed 0.1 mg/kg of Se was lower (P < 0.05) in
hens fed Se yeast, whereas from hens fed 0.3 mg/kg of
Se, it was comparable across treatments. Selenium in
egg, liver, and breast muscle was greater (P < 0.01 or
<0.05) in hens fed the greater concentration of Se
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