In our findings, increasing fasting blood glucose was the major independent variable associated
with hypertension. The overall prevalence of IFG in our study was lower than the Addis
Ababa report (21.6%) and similar with that of a study done in Morocco, 5.5% [8,20]. Moreover,
the computed prevalence of IFG differs significantly with areas of residence and age. The rate
increased modestly with age and highly among urban dwellers. Similarly, a study conducted in
South Africa showed the prevalence of IFG increased with age, with peak prevalence in the oldest
age-group. [21–23]. Impaired Fasting Glucose (IFG) is a predictor of the incidence of Type
2 diabetes and hypertension [23,24]. Epidemiological studies and path-physiological mechanisms
provide evidence to the co-existence of hypertension, IFG, and abdominal obesity, possibly
pointing towards a common genetic and environmental factor promoting the risk of CVD
[25,26]. Similarly, there is a strong association between abdominal obesity, rise in FBG, and hypertension which was also observed in our finding [27].Therefore, the persistent high prevalence
of IFG may be a possible predictor of a further increase in hypertension in this
population in the years to come [28].
 
In our findings, increasing fasting blood glucose was the major independent variable associatedwith hypertension. The overall prevalence of IFG in our study was lower than the AddisAbaba report (21.6%) and similar with that of a study done in Morocco, 5.5% [8,20]. Moreover,the computed prevalence of IFG differs significantly with areas of residence and age. The rateincreased modestly with age and highly among urban dwellers. Similarly, a study conducted inSouth Africa showed the prevalence of IFG increased with age, with peak prevalence in the oldestage-group. [21–23]. Impaired Fasting Glucose (IFG) is a predictor of the incidence of Type2 diabetes and hypertension [23,24]. Epidemiological studies and path-physiological mechanismsprovide evidence to the co-existence of hypertension, IFG, and abdominal obesity, possiblypointing towards a common genetic and environmental factor promoting the risk of CVD[25,26]. Similarly, there is a strong association between abdominal obesity, rise in FBG, and hypertension which was also observed in our finding [27].Therefore, the persistent high prevalenceof IFG may be a possible predictor of a further increase in hypertension in thispopulation in the years to come [28].
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