This finding is not in agreement with the study by Khanzode SD et al where a significant EPS potential was displayed by nimodipine. It may be hypothesized that although nimodipine inhibits dopamine activity (thus the antipsychosis), it may not modify the dopamine/acetylcholine balance significantly at the levels of basal ganglion at the doses used in this study. Furthermore nimodipine has been suggested as useful adjunctive treatment of tardive dyskinesia’s which is actually a delayed type of extra pyramidal symptom due to typical antipsychotics.