In this report, we evaluated the pattern of hypoxic damage across 2 imaging studies over a period of two weeks and correlated these finding with the clinical signs and symptoms up to 15 weeks after the injury. Our patient
remained affected by significant loss of motor control and increased extensor tone in the lower extremities; he devel- oped impaired balance and inability to ambulate that improved minimally. These findings are consistent with damage to the motor system including globus pallidus, cerebellum and frontal lobes. Despite the significant motor problems, the sensory and the non-executive cog- nitive systems of the patient remained intact. A review of the literature on the reported cases of hypoxic brain injury reveals that the predominant clinical residues after pure hypoxic injury are motor disorders, and that the motor disturbances may manifest after significant latency [7,9- 11]. For example, Feve et al. [9] have described that lesions confined to globus pallidus after a hypoxic brain insult are associated with residual axial motor distur- bances. Movement disorders have been described in young children years after the initial hypoxic brain injury.