precise pathogenesis
of the disease is uncertain, there appears to be decreased
laminar perfusion, with varying degrees of inflammation
and necrosis of the sensitive laminae. Causes include carbohydrate
overload, endotoxaemia (such as caused by gastrointestinal
disease, septic metritis and pleuritis), excessive
work, and hyperadrenocorticism (Cushing’s syndrome).
precise pathogenesisof the disease is uncertain, there appears to be decreasedlaminar perfusion, with varying degrees of inflammationand necrosis of the sensitive laminae. Causes include carbohydrateoverload, endotoxaemia (such as caused by gastrointestinaldisease, septic metritis and pleuritis), excessivework, and hyperadrenocorticism (Cushing’s syndrome).
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