On the other hand, the effects of EGCG on insulin resistance
may partially contribute to the prevention of hepatic steatosis.
Insulin resistance causes increased plasma FFA and enhances
liver uptake of fatty acids, which are then stored as triglycerides
(34). It is possible that EGCG improves insulin sensitivity and
consequently reduces the risk for hepatic steatosis. Further
studies are needed to determine the causal relationship between
insulin resistance and hepatic steatosis, and the role of EGCG in
attenuating both of these conditions.